Martin et al., 2024 - Google Patents
Role of IL-27 in Epstein–Barr virus infection revealed by IL-27RA deficiencyMartin et al., 2024
View PDF- Document ID
- 14922505635777667536
- Author
- Martin E
- Winter S
- Garcin C
- Tanita K
- Hoshino A
- Lenoir C
- Fournier B
- Migaud M
- Boutboul D
- Simonin M
- Fernandes A
- Bastard P
- Le Voyer T
- Roupie A
- Ben Ahmed Y
- Leruez-Ville M
- Burgard M
- Rao G
- Ma C
- Masson C
- Soudais C
- Picard C
- Bustamante J
- Tangye S
- Cheikh N
- Seppänen M
- Puel A
- Daly M
- Casanova J
- Neven B
- Fischer A
- Latour S
- Publication year
- Publication venue
- Nature
External Links
Snippet
Epstein–Barr virus (EBV) infection can engender severe B cell lymphoproliferative diseases,. The primary infection is often asymptomatic or causes infectious mononucleosis (IM), a self-limiting lymphoproliferative disorder. Selective vulnerability to EBV has been …
- 108010066979 Interleukin-27 0 title abstract description 226
Classifications
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/5005—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells
- G01N33/5008—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics
- G01N33/5044—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing involving human or animal cells for testing or evaluating the effect of chemical or biological compounds, e.g. drugs, cosmetics involving specific cell types
- G01N33/5047—Cells of the immune system
- G01N33/505—Cells of the immune system involving T-cells
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/53—Immunoassay; Biospecific binding assay
- G01N33/569—Immunoassay; Biospecific binding assay for micro-organisms, e.g. protozoa, bacteria, viruses
-
- G—PHYSICS
- G01—MEASURING; TESTING
- G01N—INVESTIGATING OR ANALYSING MATERIALS BY DETERMINING THEIR CHEMICAL OR PHYSICAL PROPERTIES
- G01N33/00—Investigating or analysing materials by specific methods not covered by the preceding groups
- G01N33/48—Investigating or analysing materials by specific methods not covered by the preceding groups biological material, e.g. blood, urine; Haemocytometers
- G01N33/50—Chemical analysis of biological material, e.g. blood, urine; Testing involving biospecific ligand binding methods; Immunological testing
- G01N33/53—Immunoassay; Biospecific binding assay
- G01N33/564—Immunoassay; Biospecific binding assay for pre-existing immune complex or autoimmune disease, i.e. systemic lupus erythematosus, rheumatoid arthritis, multiple sclerosis, rheumatoid factors or complement components C1-C9
-
- C—CHEMISTRY; METALLURGY
- C12—BIOCHEMISTRY; BEER; SPIRITS; WINE; VINEGAR; MICROBIOLOGY; ENZYMOLOGY; MUTATION OR GENETIC ENGINEERING
- C12N—MICRO-ORGANISMS OR ENZYMES; COMPOSITIONS THEREOF; PROPAGATING, PRESERVING OR MAINTAINING MICRO-ORGANISMS; MUTATION OR GENETIC ENGINEERING; CULTURE MEDIA
- C12N5/00—Undifferentiated human, animal or plant cells, e.g. cell lines; Tissues; Cultivation or maintenance thereof; Culture media therefor
- C12N5/06—Animal cells or tissues; Human cells or tissues ; Not used, see subgroups
- C12N5/0602—Vertebrate cells
- C12N5/0634—Cells from the blood or the immune system
- C12N5/0636—T lymphocytes
-
- A—HUMAN NECESSITIES
- A61—MEDICAL OR VETERINARY SCIENCE; HYGIENE
- A61K—PREPARATIONS FOR MEDICAL, DENTAL, OR TOILET PURPOSES
- A61K35/00—Medicinal preparations containing materials or reaction products thereof with undetermined constitution
- A61K35/12—Materials from mammals; Compositions comprising non-specified tissues or cells; Compositions comprising non-embryonic stem cells; Genetically modified cells
Similar Documents
| Publication | Publication Date | Title |
|---|---|---|
| Martin et al. | Role of IL-27 in Epstein–Barr virus infection revealed by IL-27RA deficiency | |
| Sureshchandra et al. | Single-cell profiling of T and B cell repertoires following SARS-CoV-2 mRNA vaccine | |
| Izawa et al. | Inherited CD70 deficiency in humans reveals a critical role for the CD70–CD27 pathway in immunity to Epstein-Barr virus infection | |
| Boutboul et al. | Dominant-negative IKZF1 mutations cause a T, B, and myeloid cell combined immunodeficiency | |
| Martin et al. | CTP synthase 1 deficiency in humans reveals its central role in lymphocyte proliferation | |
| Abolhassani et al. | Combined immunodeficiency and Epstein-Barr virus–induced B cell malignancy in humans with inherited CD70 deficiency | |
| Yun et al. | Human plasmacytoid dendritic cells mount a distinct antiviral response to virus-infected cells | |
| Ju et al. | Modulation of STAT‐3 in rheumatoid synovial T cells suppresses Th17 differentiation and increases the proportion of Treg cells | |
| Herrmann et al. | Analysis of co-inhibitory receptor expression in COVID-19 infection compared to acute Plasmodium falciparum malaria: LAG-3 and TIM-3 correlate with T cell activation and course of disease | |
| Pitard et al. | Long-term expansion of effector/memory Vδ2− γδ T cells is a specific blood signature of CMV infection | |
| Strauss et al. | Differential responses of human regulatory T cells (Treg) and effector T cells to rapamycin | |
| Schmitt et al. | IL-12 receptor β1 deficiency alters in vivo T follicular helper cell response in humans | |
| Ronchetti et al. | Glucocorticoid‐induced tumour necrosis factor receptor‐related protein: a key marker of functional regulatory T cells | |
| Kared et al. | Adaptive NKG2C+ CD57+ natural killer cell and Tim-3 expression during viral infections | |
| Longhi et al. | Characterization of human CD39+ Th17 cells with suppressor activity and modulation in inflammatory bowel disease | |
| Ji et al. | HIV-1 binding to CD4 on CD4+ CD25+ regulatory T cells enhances their suppressive function and induces them to home to, and accumulate in, peripheral and mucosal lymphoid tissues: an additional mechanism of immunosuppression | |
| Huygens et al. | Functional exhaustion limits CD4+ and CD8+ T-cell responses to congenital cytomegalovirus infection | |
| Ambegaonkar et al. | The differentiation in vitro of human tonsil B cells with the phenotypic and functional characteristics of T-bet+ atypical memory B cells in malaria | |
| Govender et al. | T cell perturbations persist for at least 6 months following hospitalization for COVID-19 | |
| Spoerl et al. | Upregulation of CCR4 in activated CD8+ T cells indicates enhanced lung homing in patients with severe acute SARS‐CoV‐2 infection | |
| Pillay et al. | Hematopoietic stem cell transplant effectively rescues lymphocyte differentiation and function in DOCK8-deficient patients | |
| Cooper et al. | Rapid GMP-compliant expansion of SARS-CoV-2–specific T cells from convalescent donors for use as an allogeneic cell therapy for COVID-19 | |
| Ihantola et al. | Effector T cell resistance to suppression and STAT3 signaling during the development of human type 1 diabetes | |
| WO2009036521A1 (en) | A method for identifying antigen-specific regulatory t cells | |
| Tang et al. | Characterization of age-related immune features after autologous NK cell infusion: Protocol for an open-label and randomized controlled trial |